Anxiety (restlessness, fear) is a physiological protection system for the organism. Protects the body from danger in case of a potential danger.

However, in some cases, such as chronic stress, brain cells are prone to damage since the construction, quality and concentration of neurotransmitters are impaired. As a result, people show a severe anxiety reaction to the non-existent danger as if there is a real threat. This short-term intense anxiety is called panic attack.

Norepinephrine has increased in the brain in panic attack patients, serotonin and GABA neurotransmitters have decreased. First of all, it is necessary to measure them.

Depending on the increase in noradrenaline, tachycardia, dyspnea, sweating, dry mouth, high blood pressure, tremors in the hands, fear of death, enlargement of the pupils(midriasis), increase in bowel movements and gas formation.

Lack of appetite due to decreased serotonin and also low energy, insomnia, mood imbalance, sexual reluctance occurs.

Sleep deprivation and deterioration in sleep quality occur due to decreased GABA. Panic attackers often become addicted to alcohol in order to eliminate these symptoms and to sleep. Panic attack crisis occurs when alcohol dependence is treated. The same findings emerge in the deprivation crisis of substance abuse.

Common treatment methods;

Common drug treatments are intended to correct the effects of noradrenaline, serotonin, and GABA neurotransmitters. However, these treatments do not improve the production and quality of these neurotransmitters. For this reason, treatment is not permanent. If the patient does not use the drug for a lifetime, the disease reoccurs.

Ideal treatment;

It is the treatment that permanently removes the damage in the brain cells, provide the quality and balance of the neurotransmitters, and eliminates the need for long-term use of the drug by the patient.

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